Cardiomyopathy: Difference between revisions
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Click [http://www.cardiogenetica.nl/index.php?menuID=2 '''here'''] for detailed information on various cardiomyopathy | Click [http://www.cardiogenetica.nl/index.php?menuID=2 '''here'''] for detailed information on various cardiomyopathy. | ||
{| class="wikitable" cellpadding="0" cellspacing="0" border="0" | {| class="wikitable" cellpadding="0" cellspacing="0" border="0" | ||
|+ Listed below are the main disorders and their characteristics with examples.<cite>1</cite> | |||
|- | |- | ||
!Condition | !Condition | ||
!Features | !Features | ||
!Example | !Example | ||
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!rowspan="4" valign="top"|Arrythmogene right ventricular cardiomyopathy (ARVC) | !rowspan="4" valign="top"|Arrythmogene right ventricular cardiomyopathy (ARVC) | ||
|rowspan="4" valign="top"| | |rowspan="4" valign="top"| | ||
*Fibrofatty degeneration of the RV . | *Fibrofatty degeneration of the RV. | ||
*Myocardial degeneration leads to RV dilation and poor RVF . | *Myocardial degeneration leads to RV dilation and poor RVF. | ||
*Ventricular fibrillation by slow conduction velocities, guide block and spatial variation in conduction velocity. | *Ventricular fibrillation by slow conduction velocities, guide block and spatial variation in conduction velocity. | ||
*Aneurysms of the RV free wall. | *Aneurysms of the RV free wall. | ||
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*35% symmetrical hypertrophy of the myocardium (not to be confused with aortic stenosis or hypertension). | *35% symmetrical hypertrophy of the myocardium (not to be confused with aortic stenosis or hypertension). | ||
*Small LV lumen. | *Small LV lumen. | ||
*Preserved systolic LV function ( EF normal or slightly decreased) | *Preserved systolic LV function (EF normal or slightly decreased) | ||
*Diastolic dysfunction. | *Diastolic dysfunction. | ||
*Autosomal dominant progressive deviation from nature. | *Autosomal dominant progressive deviation from nature. | ||
* | *Could be associated with sudden cardiac death due to ventricular fibrillation, an increased risk of thromboembolism. | ||
*Heart failure can be caused by the rigidity of the thickened heart muscle (diastolic heart failure), by an obstruction in the LVOT (SAM ) is associated with mitral valvular insufficiency. The course of the disease is progressive. | *Heart failure can be caused by the rigidity of the thickened heart muscle (diastolic heart failure), by an obstruction in the LVOT (SAM) and is associated with mitral valvular insufficiency. The course of the disease is progressive. | ||
*Occurs in persons 1:500-1000 | *Occurs in persons 1:500-1000 | ||
|[[Image:Asym.cmp1.jpg|400px]] | |[[Image:Asym.cmp1.jpg|400px]] | ||
|- | |- | ||
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!Apical hypertrophy | !Apical hypertrophy | ||
|- | |- | ||
|Non - compaction cardiomyopathy ( NCCMP ) | !rowspan="4" valign="top"|Non-compaction cardiomyopathy (NCCMP) | ||
|rowspan="4" valign="top"| | |||
*LV wall has a spongy appearance. | |||
Jenni criteria ( Heart 2007) . | *Jenni criteria (Heart 2007). | ||
Also called insulated non compaction of the ventricular myocardium ( INVM ) , is a rare form of congenital heart disease in which the tissue of the ventricular myocardium is constructed in terms of texture | *Also called insulated non compaction of the ventricular myocardium (INVM), it is a rare form of congenital heart disease in which the tissue of the ventricular myocardium is not well constructed in terms of texture. | ||
*After HCM DCM, it is the most common cause of primary cardiomyopathy in children. | |||
It is a congenital defect , which occurs in the 20th week of pregnancy . | *It is a congenital defect, which occurs in the 20th week of pregnancy. | ||
The condition is expressed by heart failure, arrhythmias, and an increased risk of thrombus formation. | *The condition is expressed by heart failure, arrhythmias, and an increased risk of thrombus formation. | ||
The disorder often manifests itself later in life and has a high mortality rate due to heart failure and arrhythmias . | *The disorder often manifests itself later in life and has a high mortality rate due to heart failure and arrhythmias. | ||
|[[Image:NCCMP02.jpg|400px]] | |||
NCCMP with crypts and apical midventriculair | |- | ||
!NCCMP with crypts and apical midventriculair | |||
NCCCMP on PSax | |- | ||
|Video | |||
|- | |||
!NCCCMP on PSax | |||
Stiffened myocardium. | |- | ||
This form comes after at least 1 and is usually associated with storage diseases , such as sarcoidosis , amyloidosis , and the like. | !rowspan="2" valign="top"|Restrictive cardiomyopathy | ||
Preserved systolic LVF . LV is not dilated . | |rowspan="2" valign="top"| | ||
Diastolic dysfunction ( see fig.) | *Stiffened myocardium. | ||
LV and RV may be hypertrophied . | *This form comes after at least 1 and is usually associated with storage diseases, such as sarcoidosis, amyloidosis, and the like. | ||
Dilated atria and VCI . | *Preserved systolic LVF. LV is not dilated. | ||
Pulmonary hypertension . | *Diastolic dysfunction (see fig.) | ||
Myocardial echo during and amyloidosis speckled | *LV and RV may be hypertrophied. | ||
*Dilated atria and VCI. | |||
Abnormally low É in restrictive | *Pulmonary hypertension. | ||
Tako- tsubo cardiomyopathy | *Myocardial echo during and amyloidosis speckled | ||
|[[Image:Restrcmp.jpg|400px]] | |||
Apical ballooning , | |- | ||
!Abnormally low É in restrictive cardiomyopathy | |||
Is more common in women than in men , the average age of 62 to 75 years . | |- | ||
!rowspan="2" valign="top"|Tako-tsubo cardiomyopathy | |||
Also called "broken heart syndrome" or " Stress CMP " | |rowspan="2" valign="top"| | ||
LV | *Takotsubo is named after the ceramic pots used to trap octopus in Japan. | ||
*Apical ballooning, akinetic of the apex. This gives the LV the octupus trap shape. | |||
Apical ballooning | *Manifests itself as an acute myocardial infarction with ST elevations, however, no significant coronary artery disease. | ||
*Is more common in women than in men, the average age of 62 to 75 years. | |||
*Stress induced, is triggered by an acute illness or intense emotional or physical stress | |||
*Also called "broken heart syndrome" or "Stress CMP". | |||
*LV normalizes in a few days to several weeks. | |||
|[[Image:TakoTsubo01.jpg|400px]] | |||
|- | |||
!Apical ballooning | |||
|} | |} | ||
==References== | ==References== | ||
<biblio> | <biblio> | ||
#1 [http://www.uptodate.com/contents/echocardiographic-recognition-of-cardiomyopathies?source=search_result&search=cardiomyopathy+echo&selectedTitle=1~150| Echocardiographic recognition of cardiomyopathies] | #1 [http://www.uptodate.com/contents/echocardiographic-recognition-of-cardiomyopathies?source=search_result&search=cardiomyopathy+echo&selectedTitle=1~150| Echocardiographic | ||
#2 recognition of cardiomyopathies] | |||
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</biblio> | </biblio> |
Latest revision as of 04:33, 15 June 2021
Cardiomyopathy (CMP) is a collective term for various diseases of the heart muscle (myocardium). For various reasons, the function of the myocardium decreased (see table). The different variants of a CMP are generally classified on the basis of echocardiographic characteristics.
LV function decline in most common cardiomyopathy | ||
---|---|---|
Systolic function | Diastolic Function | |
Dilated CMP | ↓ | =/↓ |
Hypertrophic CMP | ↑ | ↓ |
Restrictive CMP | = | ↓ |
Click here for detailed information on various cardiomyopathy.
Condition | Features | Example |
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Arrythmogene right ventricular cardiomyopathy (ARVC) |
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Video |
Echodense RV free wall for suspected ARVC | ||
Decreased RV strain in ARVC | ||
Dilated cardiomyopathy (DCM) |
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Dilated LV on AP4CH | ||
Dilated LV on PLAX | ||
EPSS is a useful measurement to follow up DCM | ||
Hypertrophic cardiomyopathy (HCM) |
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Asymmetric hypertrophy | ||
Symmetrical hypertrophy | ||
Apical hypertrophy | ||
Non-compaction cardiomyopathy (NCCMP) |
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NCCMP with crypts and apical midventriculair | ||
Video | ||
NCCCMP on PSax | ||
Restrictive cardiomyopathy |
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Abnormally low É in restrictive cardiomyopathy | ||
Tako-tsubo cardiomyopathy |
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Apical ballooning |
References
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recognition of cardiomyopathies]