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Cardiomyopathy: Difference between revisions
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!Apical hypertrophy | !Apical hypertrophy | ||
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|Non - compaction cardiomyopathy (NCCMP) | !rowspan="4" valign="top"|Non-compaction cardiomyopathy (NCCMP) | ||
|rowspan="4" valign="top"| | |||
*LV wall has a spongy appearance. | |||
Jenni criteria (Heart 2007). | *Jenni criteria (Heart 2007). | ||
Also called insulated non compaction of the ventricular myocardium (INVM), is a rare form of congenital heart disease in which the tissue of the ventricular myocardium is constructed in terms of texture | *Also called insulated non compaction of the ventricular myocardium (INVM), it is a rare form of congenital heart disease in which the tissue of the ventricular myocardium is not well constructed in terms of texture. | ||
*After HCM DCM, it is the most common cause of primary cardiomyopathy in children. | |||
It is a congenital defect, which occurs in the 20th week of pregnancy. | *It is a congenital defect, which occurs in the 20th week of pregnancy. | ||
The condition is expressed by heart failure, arrhythmias, and an increased risk of thrombus formation. | *The condition is expressed by heart failure, arrhythmias, and an increased risk of thrombus formation. | ||
The disorder often manifests itself later in life and has a high mortality rate due to heart failure and arrhythmias. | *The disorder often manifests itself later in life and has a high mortality rate due to heart failure and arrhythmias. | ||
|[[Image:NCCMP02.jpg|400px]] | |||
NCCMP with crypts and apical midventriculair | |- | ||
!NCCMP with crypts and apical midventriculair | |||
NCCCMP on PSax | |- | ||
|Video | |||
|- | |||
!NCCCMP on PSax | |||
Stiffened myocardium. | |- | ||
This form comes after at least 1 and is usually associated with storage diseases, such as sarcoidosis, amyloidosis, and the like. | !rowspan="2" valign="top"|Restrictive cardiomyopathy | ||
Preserved systolic LVF. LV is not dilated. | |rowspan="2" valign="top"| | ||
Diastolic dysfunction (see fig.) | *Stiffened myocardium. | ||
LV and RV may be hypertrophied. | *This form comes after at least 1 and is usually associated with storage diseases, such as sarcoidosis, amyloidosis, and the like. | ||
Dilated atria and VCI. | *Preserved systolic LVF. LV is not dilated. | ||
Pulmonary hypertension. | *Diastolic dysfunction (see fig.) | ||
Myocardial echo during and amyloidosis speckled | *LV and RV may be hypertrophied. | ||
*Dilated atria and VCI. | |||
Abnormally low É in restrictive | *Pulmonary hypertension. | ||
Tako- tsubo cardiomyopathy | *Myocardial echo during and amyloidosis speckled | ||
Tako- tsubo are octopus lure often used in Japan. | |[[Image:Restrcmp.jpg|400px]] | ||
|- | |||
!Abnormally low É in restrictive cardiomyopathy | |||
|- | |||
!rowspan="2" valign="top"|Tako-tsubo cardiomyopathy | |||
|rowspan="2" valign="top"| | |||
*Tako- tsubo are octopus lure often used in Japan. | |||
Apical ballooning, akinesia of the apex. This gives the LV configuration octopus lure. | Apical ballooning, akinesia of the apex. This gives the LV configuration octopus lure. | ||
manifests itself as an acute myocardial infarction with ST elevations, however, no significant coronary artery disease. | manifests itself as an acute myocardial infarction with ST elevations, however, no significant coronary artery disease. | ||
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Also called "broken heart syndrome" or " Stress CMP " called. | Also called "broken heart syndrome" or " Stress CMP " called. | ||
LV normalized to several weeks in few days. | LV normalized to several weeks in few days. | ||
|[[Image:TakoTsubo01.jpg|400px]] | |||
Apical ballooning | |- | ||
!Apical ballooning | |||
|} | |} | ||